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The Drug-Induced Respiratory Disease Website

(Fr: bronchospasme, asthme). Including bronchospasm, bronchoconstriction, de novo subacute asthma, asthma attack and deterioration or exacerbation of preexisting/underlying asthma. Can be sudden and very severe, causing asphyxia followed by irreversible hypoxic brain damage (see also under IVf). NSAIDs, ß-blockers and antibiotics are the main causal agents. Patients with a background of atopy or asthma are particularly at risk. In a few individuals, the asthma may persist despite cessation of exposure (PMID 45308). Healthcare workers can be involved when handling drugs (PMID 3612323). Workers in the drug-manufacturing (e.g. antibiotics) industry can develop bronchospasm or asthma as a manifestation of drug-induced occupational disease (PMID 16436155, 21603168, 30075340)

Publications

Marcus EL, Hush A, Atrash H, Shibli R, Heyman SN

Life-threatening bronchospasm induced by an angiotensin-converting enzyme inhibitor in a chronically ventilated patient: Diagnostic pitfalls and literature review.

Respirology case reports 2023 Oct;11;e01224 — 2023 Oct — e01224

Yilmaz ?, Türk M, Baran Ketencio?lu B, Çetinkaya Z, Tutar N, Oymak FS, Gülmez ?, Adkinson NF

The presence of underlying asthma should be investigated in patients diagnosed with ACE inhibitor induced cough.

The clinical respiratory journal 2020 Jan 04;; — 2020 Jan 04

Overlack A

ACE inhibitor-induced cough and bronchospasm. Incidence, mechanisms and management.

Drug safety 1996 Jul;15;72-8 — 1996 Jul — 72-8

Inman WH, Wilton L, Pearce G, Mann RD

Angiotensin converting enzyme inhibitors and asthma.

BMJ (Clinical research ed.) 1994 Feb 26;308;593-4 — 1994 Feb 26 — 593-4

Popa V

Captopril-related (and -induced?) asthma.

The American review of respiratory disease 1987 Oct;136;999-1000 — 1987 Oct — 999-1000